News from the University of Yemen An American research team concluded that a supply of allogenegenolone (ALLO) during pregnancy may lead to the fetus being affected by autism spectrum disorders, according to the news eye.
The hormone "alopergannolone" is made by the placenta at the end of pregnancy, a powerful neurotransmitter, to the extent that disruption of the supply line presents the fetus to the autism spectrum disorder, according to a study presented Saturday at the annual meeting of the American Academy of Pediatrics in America.
According to the Centers for Disease Control and Prevention in America, about 1 in 10 children are prematurely born, making birth prematurely a significant risk factor that disconnects the supply of the hormone that reaches the fetus through the placenta.
The placenta is a flat circular organ that communicates with the fetus through the umbilical cord, through which embryos, oxygen and glucose are transferred to the fetus, as well as the fetus with the hormone alopergenolone, a progesterone derivative, to prepare the embryonic brain that develops into ectopic life.
"The hormone is made by the placenta at the end of pregnancy," says Anna Benn, a brain development researcher and principal investigator. "When children are born prematurely, their supply stops, it happens at the same time as the cerebellum – a central area of brain coordination. – Usually a huge growth boom.
"We have demonstrated using an experimental model using mice that the loss of the alopergananol hormone changes the development of the cerebellum, including the development of white matter," she said.
And white matter, one of the two components of the central nervous system, and the development of the cerebellum in the brain, especially after the birth of children, so that the association of change in the placenta during pregnancy with long-term effects on brain development is a particularly prominent result, as confirmed by the researcher.
The team confirmed this by deleting the gene encoding the placental enzyme responsible for placental production in the experimental model, comparing it to another model in which it was not removed, and performing the complete EEG analysis and expression of the two groups.
"This finding may help us in the future to provide early treatment for early problems that can be dealt with," she said.
Study: Hypromagnolone deficiency affects the fetus by autism, news we presented to you through our site.
This study was imported from research: a deficiency of hypromagnolone affects the fetus by autism, the main source of which is the Arab site of Yemen.
We do not assume in Yemen any responsibility for research content: a deficiency of the hormone "alopergannolone" affects the fetus by autism.